Nathan Young, PhD
Nathan Young, PhD
Orthopaedic Trauma Institute - Young Lab
Orthopaedic Trauma Institute at Zuckerberg San Francisco General
Education
| Postdoctoral Studies | University of California, San Francisco | 2007 | |
| Postdoctoral Studies | Stanford University | 2006 | |
| Postdoctoral Studies | University of Calgary | 2004 | |
| Ph.D. | Harvard University | 2003 | |
| B.A. | New College of Florida | 1995 |
Publications
- Young NM. Integrating "Evo" and "Devo": The Limb as Model Structure. Integr Comp Biol. 2017 12 01; 57(6):1293-1302. PMID: 28992180
- Young NM. Function, ontogeny and canalization of shape variance in the primate scapula. J Anat. 2006 Nov; 209(5):623-36. PMID: 17062020
- Steiper ME, Young NM. Primate molecular divergence dates. Mol Phylogenet Evol. 2006 Nov; 41(2):384-94. PMID: 16815047
- Young N. Modularity and integration in the hominoid scapula. J Exp Zool B Mol Dev Evol. 2004 May 15; 302(3):226-40. PMID: 15211684
Grants & Awards
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The developmental genetic basis for evolutionary variation in the hominin shoulder
July 1, 2015 - June 30, 2018
NSF BCS-1518596
Role: Co-Investigator
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A Molecular Investigation of Growth and Shape Variation in Cleft-Lip Etiology
September 1, 2007 - August 31, 2009
NIH/NIDCR F32DE018596
Role: Principal Investigator
Membership & Committees
About Nathan Young, PhD
I address basic biomedical research questions through the lens of evolution, utilizing variation, functional compromise, and historical constraint as fundamental explanatory principles. My research program combines classical embryology, modern genetic tools with advanced methods for quantifying and comparing phenotypes at a range of scales. When combined with mechanistic insights from experimental systems, this approach yields significant insights into the generation of individual phenotypes, both normal and abnormal. This approach has significance for understanding not only the processes that contribute to evolutionary diversity, but also the individual phenotypic differences found within species and among individuals, including dysmorphologies associated with human disease states. My research therefore includes the study of normal mechanisms of development as well as the etiology of congenital developmental defects, and is strongly relevant to longstanding goals of providing personalized and predictive medicine.