Nathan Young, PhD

Nathan Young, PhD
Orthopaedic Trauma Institute - Young Lab
2550 23rd Street
San Francisco CA 94110
628-206-8812
San Francisco CA 94110
Orthopaedic Trauma Institute at Zuckerberg San Francisco General
Publications
- Young NM. Function, ontogeny and canalization of shape variance in the primate scapula. J Anat. 2006 Nov; 209(5):623-36. PMID: 17062020
- Steiper ME, Young NM. Primate molecular divergence dates. Mol Phylogenet Evol. 2006 Nov; 41(2):384-94. PMID: 16815047
- Young N. Modularity and integration in the hominoid scapula. J Exp Zool B Mol Dev Evol. 2004 May 15; 302(3):226-40. PMID: 15211684
- Young NM, MacLatchy L. The phylogenetic position of Morotopithecus. J Hum Evol. 2004 Feb; 46(2):163-84. PMID: 14871561
Grants & Awards
- A Predictive Developmental Morphospace Model of Cleft Lip
2020-09-03 - 2021-08-31
NIH R56DE029124
Role: Principal Investigator
- The developmental genetic basis for evolutionary variation in the hominin shoulder
2015-07-01 - 2018-06-30
NSF BCS-1518596
Role: Co-Investigator
- A Molecular Investigation of Growth and Shape Variation in Cleft-Lip Etiology
2007-09-01 - 2009-08-31
NIH F32DE018596
Role: Principal Investigator
Education
Postdoctoral Studies | University of California, San Francisco | 2007 | |
Postdoctoral Studies | Stanford University | 2006 | |
Postdoctoral Studies | University of Calgary | 2004 | |
Ph.D. | Harvard University | 2003 | |
B.A. | New College of Florida | 1995 |
About Nathan Young, PhD
My research utilizes the concepts of variation, functional compromise, and historical constraint as fundamental explanatory principles to address basic biomedical questions through the lens of evolution. My lab combines classical embryology in a range of model and non-model systems with modern genetic tools and advanced methods for quantifying and comparing phenotypes at a range of scales. This approach yields significant insights into not only the processes that contribute to evolutionary diversity among species, but also how individual differences ranging from normal to disease states are generated. My research therefore investigates both normal mechanisms of development as well as the etiology of structural birth defects, and is relevant to longstanding goals of providing personalized and predictive medicine.