Hubert Kim, MD, PhD
Hubert Kim, MD, PhD
Education
| M.D. | Stanford University | 1993 | |
| Residency | University of California, San Francisco |
Publications
- Crawford MD, Kim HT. New-onset synovial chondromatosis after total knee arthroplasty. J Arthroplasty. 2013 Feb; 28(2):375.e1-4. PMID: 22704224
- Piper SL, Kim HT. Thermal stress potentiates bupivacaine chondrotoxicity. Arthroscopy. 2012 Sep; 28(9):1246-1254.e1. PMID: 22579775
- Laron D, Samagh SP, Liu X, Kim HT, Feeley BT. Muscle degeneration in rotator cuff tears. J Shoulder Elbow Surg. 2012 Feb; 21(2):164-74. PMID: 22244059
- Piper SL, Kramer JD, Kim HT, Feeley BT. Effects of local anesthetics on articular cartilage. Am J Sports Med. 2011 Oct; 39(10):2245-53. PMID: 21515808
- Liu X, Manzano G, Kim HT, Feeley BT. A rat model of massive rotator cuff tears. J Orthop Res. 2011 Apr; 29(4):588-95. PMID: 20949443
Grants & Awards
Membership & Committees
About Hubert Kim, MD, PhD
Hubert Kim graduated with Honors and with Distinction from Stanford University in 1986 with a B.S. in Biological Sciences. He graduated from the Stanford University School of Medicine in 1993, receiving both his MD degree and a PhD degree in Cancer Biology as a Howard Hughes Pre-doctoral fellow. He completed his residency in Orthopaedic Surgery at the University of California San Francisco in 1998.
Upon completion of his residency, Dr. Kim joined the faculty at the University of California, San Francisco and was appointed as a Staff Physician at the San Francisco VA Medical Center. In 2001 he joined the practice at the San Francisco General Hospital Division of Orthopaedic Surgery as an Attending Physician specializing in joint replacement and adult reconstruction.
Dr. Kim's research focus is the molecular and cellular mechanisms responsible for secondary injury cascades that are set in motion after mechanical trauma. He is particularly interested in tissues that have a very limited capacity for healing and regeneration where preservation of existing cells and tissue may be of particular clinical significance. Specifically, he is investigating the mechanisms involved in programmed cell death following acute trauma involving articular cartilage and the spinal cord. His goal is to eventually apply lessons learned in the laboratory to the design of better treatments for patients.
